ios - 解析 XML - 捕获的字符串被前面截断。 swift

Question

我对 xml 文档的解析工作正常，但我注意到某些标签(“AbstractText”)之间的文本随机被截断，我不知道是什么。同样，这只出现在这里和那里，并不总是这个标签的情况。我的解析代码如下，也是截断文本的示例。

 var abstractBool = false
 var abstract = ""

 func parser(_ parser: XMLParser, didStartElement elementName: String, namespaceURI: String?, qualifiedName qName: String?, attributes attributeDict: [String : String] = [:]) {
      switch elementName {
       case "AbstractText":
             abstractBool = true
      }
}


 func parser(_ parser: XMLParser, foundCharacters string: String) {
       if abstractBool{
          abstract = string
       }
 }


 func parser(_ parser: XMLParser, didEndElement elementName: String, 
 namespaceURI: String?, qualifiedName qName: String?) {
      switch elementName {
           case "AbstractText":
           abstractBool = false
      }
 }

来自远程服务器的原始 XML:

 <Abstract>
 <AbstractText>
 Protein kinase C (PKC) has been shown to activate the mammalian target of 
 rapamycin complex 1 (mTORC1) signaling pathway, a central hub in the 
 regulation of cell metabolism, growth and proliferation. However, the 
 mechanisms by which PKCs activate mTORC1 are still ambiguous. Our previous 
 study revealed that activation of classical PKCs (cPKC) results in the 
 perinuclear accumulation of cPKC and phospholipase D2 (PLD2) in recycling 
 endosomes in a PLD2-dependent manner. Here, we report that mTORC1 activation 
 by phorbol 12,13-myristate acetate (PMA) requires both classic, cPKC, and 
 novel PKC (nPKC) isoforms, specifically PKCη, acting through distinct 
 pathways. The translocation of mTOR to perinuclear lysosomes was detected 
 after treatment of PKC activators, which was not colocalized with PKCα- or 
 RAB11-positive endosomes and was not inhibited by PLD inhibitors. We found 
 that PKCη inhibition by siRNA or bisindolylmaleimide I effectively decreased 
 mTOR accumulation in lysosomes and its activity. Also, we identified that 
 PKCη plays a role upstream of the v-ATPase/Ragulator/Rag pathway in response 
 to PMA. These data provides a spatial aspect to the regulation of mTORC1 by 
 sustained activation of PKC, requiring co-ordinated activation of two 
 distinct elements, the perinuclear accumulation of cPKC- and PLD-containing 
 endosomes and the nPKC-dependent translation of of mTOR in the perinuclear 
 lysosomes. The close proximity of these two distinct compartments shown in 
 this study suggests the possibility that transcompartment signaling may be a 
 factor in the regulation of mTORC1 activity and also underscores the 
 importance of PKCη as a potential therapeutic target of mTORC-related 
 disorders.
 </AbstractText>
 </Abstract>

我能够提取的是字符串的以下前端截断部分:

 scompartment signaling may be a factor in the regulation of mTORC1 activity 
 and also underscores the importance of PKCη as a potential therapeutic target 
 of mTORC-related disorders.

Answer 1

这是正常行为。给定部分字符串，您必须将它们连接起来，最终结果由调用 didStartElement 和 didEndElement 期间获得的所有字符串的连接给出。请务必将当前元素保存在一个变量中，以便在 foundCharacters 中您知道部分字符串所属的位置。

var abstractBool = false
var abstract = ""

func parser(_ parser: XMLParser, didStartElement elementName: String, namespaceURI: String?, qualifiedName qName: String?, attributes attributeDict: [String : String] = [:]) {
      switch elementName {
       case "AbstractText":
             abstractBool = true
             abstract = "" // if you read an XML document with multiple
                           // AbstractText elements, you need to reset 
                           // the variable
      }
}


 func parser(_ parser: XMLParser, foundCharacters string: String) {
     if abstractBool{
        abstract.append(string)
     }
 }


 func parser(_ parser: XMLParser, didEndElement elementName: String, 
 namespaceURI: String?, qualifiedName qName: String?) {
     switch elementName {
         case "AbstractText":
         abstractBool = false
         // Here the variable 'abstract' contains the full text
     }
 }